Dr. Nasrin Mehranfard | Electrophysiology | Best Researcher Award

Dr. Nasrin Mehranfard, Celluar and Molecular Medicine Research Institute, Iran

Dr. Nasrin Mehranfard is a distinguished researcher and academic from Khuzestan, Iran, with a Ph.D. in Physiology from Shahid Beheshti University of Medical Sciences, Tehran. Her research focuses on neurodegenerative disorders, ion channels, and synaptic plasticity. Dr. Mehranfard has earned significant recognition, including second rank in the Ph.D. General Entrance Exam for Physiology in Iran.She has actively participated in numerous congresses and workshops, sharing her expertise in neuroscience and electrophysiology. Her prolific contributions include over 30 publications on topics such as neuroprotection, diabetes-related neuronal changes, and the therapeutic effects of nutraceuticals. Key publications appear in prestigious journals like Pflugers Archiv and Heliyon. Dr. Mehranfard continues to advance the understanding of neurological health and disorders through her innovative research.

Publication Profile

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πŸŽ“ Academic Background

Dr. Nasrin Mehranfard is a distinguished scholar with a robust academic foundation in healthcare and physiology. She earned her B.Sc. in Midwifery from Shiraz University of Medical Sciences, Iran, showcasing her commitment to maternal and neonatal care. She further pursued an M.Sc. in Physiology at the same esteemed institution, deepening her understanding of human biological systems. Dr. Mehranfard then completed her Ph.D. in Physiology at Shahid Beheshti University of Medical Sciences in Tehran, Iran, where she honed her expertise in advanced physiological research. Her academic journey reflects a dedication to advancing health sciences and improving medical education. πŸŽ“πŸ“šβœ¨

 

πŸ”¬ Research Interests

Dr. Nasrin Mehranfard’s research focuses on advancing understanding in the field of neuroscience. Her primary interests include exploring neurodegenerative disorders 🧠, aiming to uncover mechanisms and potential treatments for conditions affecting the nervous system. She is particularly intrigued by the role of ion channels ⚑ in cellular communication and their impact on neural functionality. Additionally, Dr. Mehranfard investigates synaptic plasticity πŸ”—, delving into how neural connections adapt and reorganize in response to stimuli. Her work contributes to a deeper understanding of brain health and offers potential insights into therapeutic interventions for neurological diseases. βœ¨πŸ“Š

 

πŸ“š Teaching Experience

Dr. Nasrin Mehranfard has extensive experience in teaching physiology across prestigious institutions in Iran. She has taught medical physiology πŸ₯ at Arak University of Medical Sciences, Khomein Faculty, and physiology πŸ“– at Jahade Daneshgahee, affiliated with Tehran University. Her expertise extends to electrophysiology studies ⚑, taught at Tehran University of Medical Sciences. She has also contributed to the academic growth of students at Ghom University of Medical Sciences, Behbahan Azad University of Medical Sciences, and the University of Shiraz Nursery and Midwifery 🩺. Dr. Mehranfard’s dedication to education has greatly enriched the field of medical sciences. πŸŒŸπŸ‘©β€πŸ«

 

🧠 Research Focus

Dr. Nasrin Mehranfard’s research focuses on neuroscience 🧬, particularly in neurodegenerative disorders and neurodevelopmental alterations. Her work explores ion channels ⚑, synaptic plasticity, and the mechanisms of epilepsy, anxiety, and depression. Dr. Mehranfard has also investigated the roles of norepinephrine in neurodevelopment, neuroinflammation, and stress-related behavioral disorders πŸ§˜β€β™‚οΈ. She studies electrophysiological properties and interventions for neuroprotection, such as calcium channel blockers and natural compounds like vitamin C, honey, and naringenin 🌿. Her contributions extend to understanding the effects of early-life stress, maternal separation, and vagal nerve stimulation, positioning her as a key figure in translational neuroscience research. 🌟

 

Publication Top Notes

  • πŸ“– Anxiety and hippocampal neuronal activity: Relationship and potential mechanisms – Cited by 86, 2022
  • πŸ“– Norepinephrine, neurodevelopment and behavior – Cited by 69, 2020
  • πŸ“– Epilepsy-associated alterations in hippocampal excitability – Cited by 58, 2017
  • πŸ“– Mechanisms underlying anticonvulsant and proconvulsant actions of norepinephrine – Cited by 34, 2018
  • πŸ“– Prenatal stress and increased susceptibility to anxiety-like behaviors: role of neuroinflammation and balance between GABAergic and glutamatergic transmission – Cited by 27, 2021
  • πŸ“– Preventing effect of L-type calcium channel blockade on electrophysiological alterations in dentate gyrus granule cells induced by entorhinal amyloid pathology – Cited by 27, 2015
  • πŸ“– Long-term increases in BK potassium channel underlie increased action potential firing in dentate granule neurons following pilocarpine-induced status epilepticus in rats – Cited by 22, 2015
  • πŸ“– Calcium channel blockade attenuates abnormal synaptic transmission in the dentate gyrus elicited by entorhinal amyloidopathy – Cited by 17, 2016
  • πŸ“– The effect of paxilline on early alterations of electrophysiological properties of dentate gyrus granule cells in pilocarpine-treated rats – Cited by 14, 2014
  • πŸ“– Vitamin C protects against chronic social isolation stress-induced weight gain and depressive-like behavior in adult male rats – Cited by 11, 2020
  • πŸ“– Long-term decreases in the expression of calcineurin and GABAa receptors induced by early maternal separation are associated with increased anxiety-like behavior in adult male rats – Cited by 10, 2021
  • πŸ“– Honey protects against chronic unpredictable mild stress induced-intestinal barrier disintegration and hepatic inflammation – Cited by 10, 2020
  • πŸ“– Decrease of high voltage Ca2+ currents in the dentate gyrus granule cells by entorhinal amyloidopathy is reversed by calcium channel blockade – Cited by 10, 2017
  • πŸ“– Vagal nerve stimulation for refractory epilepsy: a brief – Cited by 9, 2016
  • πŸ“– Chronic administration of ghrelin regulates plasma glucose and normalizes insulin levels following fasting hyperglycemia and hyperinsulinemia – Cited by 9, 2015

 

 

 

Nasrin Mehranfard | Electrophysiology | Best Researcher Award

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